Diabin+

Diabin+Eating too many of today's overly processed foods strains your body's proper control of blood sugar levels and often leads to insulin insufficiency and erratic secretion of insulin - precursors to diabetes.

The ingredients in this formula support the body's ability to naturally:

  • Help use insulin more efficiently
  • Help regulate proper glucose metabolism
  • Help increase insulin sensitivity within the circulatory system
  • Help rebuild damaged pancreatic tissues

 

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purchase Order # 669 - Diabin+ $26.95

 

DIABIN+ is a Scientific Formulation of Natural Ingredients Including:

L-Alanine: Most highly concentrated amino acid released by muscle groups, functioning as a major energy source in the body; an important participant and regulator in glucose metabolism.

BCAAs: L-Leucine, L-Valine & L-Isoleucine: Specific branched chain amino acids which act to increase insulin sensitivity within the circulatory system and increase appropriate fatty acid synthesis; may also help to protect against hypertension and cardiovascular problems now known to be associated with the onset of diabetes.

Radix Trichosanthis: Helps the body naturally use fatty substances, providing provisions for energy which puts less pressure on other metabolic pathways and stores (ie, insulin-glycogen-glucose).

Fenugreek Seeds (Trigonella Foenum-Graecum): Contains an alkoloid which demonstrates hypoglycemic activity, and found in studies to exert cholesterol-lowering and blood sugar lowering effects.

Prickly Pear (Opuntia spp.): Shown to lower blood glucose and insulin levels in diabetics; believed to aid the body's ability to effectively monitor insulin levels by moving glucose from blood into body cells.

ChromeMate® GTF: A thrice patented safe chromium source, proven to assist the body in controlling insulin production and use, and more active than chromium picolinate.

Reishi Mushroom: Contains phytomedicinals demonstrated to lower blood glucose levels in diabetics.

Vitamins, Micro-Nutritionals & More: Further supports the rebalance and maintenance of the body's metabolism of fats, carbohydrates and protein, aiding in the proper management of blood glucose levels.

Research shows that when the body has optimal nutrition available that is specific to pancreatic and Immune System support, plus the electromagnetic directives to act on that nutritional input, it can manage insulin production, output and use more efficiently, and can rebuild pancreatic tissues and beta cell formations damaged by a diet disproportionately high in sugars. DIABIN+ provides this nutritional support

Sometimes we tend to eat too many simple carbohydrates (sugar, ice cream, crackers and cookies etc.) which tends to put a strain on the body's ongoing ability to deal with such overly refined foods. Our body, although miraculous, can wear out when asked to live off of a long- term compromised, overly processed diet.

And when our diets have become compromised, we usually have various problems including suffering from the "sugar blues," those deep pockets of low energy reserves that we may feel after a meal (insufficiently balanced with protein and long chain carbohydrates) when we tend to get sleepy and look for the nearest couch.

Unfortunately, such strain over time can lead to serious problems of Insulin insufficiency and erratic secretion, pancreatic disease, hormonal imbalance and Insulin receptor abnormalities, all of which may culminate into diabetes mellitus, unless the body can be rebalanced and restored through the proper diet.

Diabin+ is a special Scientifically Advanced Pancreatic Nutraceutical Dietary Supplement formulated to provide extra nutritional support for maintaining appropriate blood sugar levels within the circulatory system, whether hypo- or hyperglycemic (prediabetic), or diabetic, as well as to help the body maintain appropriate Insulin sensitivity.

Advanced Nutraceuticals, with the inclusion of ChromeMate GTF (a thrice patented safe chromium source proven to assist the body in controlling Insulin production and use), are particular and specific for the nutritional support of diabetes mellitus and diabetic ulcers, pancreatic disease and other related glandular disorders. The micro-nutritionals within Diabin+ seek to aid and support the body in its effort to rebuild and heal pancreatic tissues, ß-cell formations, control Insulin management, output and use while subsequently decreasing the amount of sugar in the urine.

Indeed, diabetes mellitus is the most common of any serious metabolic disease in humans, and perhaps more than any other disease, is closely associated with diet. Moreover, diabetic populations are significantly higher where western lifestyle and diet habits dominate rather than those cultures consuming a more "primitive" diet. Four percent of America now suffers with diabetes; 90% of these are Type II and the remaining 10% are Type I. The prevalence of diabetes is rising. It's now the 7th major cause of death in the U.S. and it's thought that this will double every 10-15 years, with an estimated 6-10% increase per year.

Diabetes mellitus is a chronic disorder of carbohydrate, fat and protein metabolism, generally characterized by fasting elevations of blood glucose levels and erratic Insulin management production, with subsequent increased chances for developing atherosclerosis, kidney disease and loss of nerve function.

Insulin is a peptide hormone secreted by the ß-cells of the islets of Langerhans of the pancreas in response to an elevation in blood glucose or other secretagogues. It plays a crucial role in glucose homeostasis by regulating the uptake and metabolism of glucose by peripheral tissues and the production and storage of glucose by the liver. Insulin also regulates the metabolism of lipids and proteins, the synthesis of nucleic acids and the expression of certain genes. In some cells, and perhaps in fetal life, Insulin also has a less well-defined role as a growth factor.

Diabetes, Type I is an Insulin-dependent diabetes (IDDM), now known to be a T-cell mediated autoimmune disease specifically targeting the pancreatic beta cells, a deficiency strongly correlated to a hereditary predisposition to injury or destruction of pancreatic ß-cells which produce and secrete Insulin, and the lack of the respective tissue regenerative power of those cells. The ß-cell insufficiency and destruction is generally caused by chemical-pH imbalances and viral or antibody damage such as that caused by inflammatory cytokines, particularly those produced by Th2-type lymphocytes, which are hypothesized to play a major role in the pathogenesis of all autoimmune diseases, including diabetes of this type, susceptible to individuals at an early age - usually childhood onset.

Diabetes, Type II is a non-Insulin dependent diabetes (NIDDM), being a disorder of glucose homeostasis characterized by hyperglycemia, peripheral Insulin resistance, impaired hepatic glucose metabolism, and diminished glucose-dependent secretion of Insulin from pancreatic ß-cells. This latter defect may lie in the glucose signaling pathway in ß-cells involving metabolically regulated Potassium channels which are the targets of sulphonylurea drugs commonly used in the treatment of NIDDM. Type II is characterized by Insulin insensitivity evidenced by typically high levels of circulating Insulin and the reversibility of blood sugar elevation by dietary changes and/or weight loss sufficient to restore Insulin sensitivity. Low GTF chromium levels are a major determinant of Insulin insensitivity, and obesity is yet another significant factor; onset is generally diet related and usually occurs later in life.

Under a physician's care, Diabin+ is safe and effective for those experiencing Diabetes Type I and Type II. It should not ever be considered a replacement for medical supervision and treatment. This formula is a special Advanced Nutraceutical medicine which supplements the body's natural ability to use Insulin more efficiently in an attempt to avoid the problems listed above, as well as to use other energy sources when sugar-energies are erratic and are at a low.

Nutraceuticals within Diabin+ include a number of herbal phytomedicinals believed to lower blood glucose levels, and quintessential Branched Chain Amino Acids (BCAAs), which act to increase Insulin sensitivity within the circulatory system, increase appropriate fatty acid synthesis and may be indirectly involved with interleukin and interferon secretion by lymph cells. Indeed, recently BCAAs and other large neutral Amino Acids have been found to be beneficial when elevated within the blood plasma of diabetic individuals. BCAAs may also help to protect against hypertension and cardiovascular problems now known to be associated with diabetic onset.

The Prickly Pear (Opuntia steptacantha, commonly known as "Nopal" in Mexico) can be found in Diabin+ as well. In a study reported in Diabetes Care and later in the Science News, Vol. 133, No.4, January 1988, this particular desert cactus was shown to lower blood glucose and Insulin levels in diabetics, the authors believing that the Prickly Pear treatment may improve the ability of Insulin to efficiently stimulate the movement of glucose from the blood into body cells.

Diabin+ is a safe and effective formulation which is further strengthened with the addition of Vaxa TriCardia+ (containing 32-Free Form Amino Acids) and Systemex, a Lactose-free Nutraceutical Meal Replacement Drink containing Casein and Colostrum.

Indeed, newly published research indicates that there is now good and compelling reason to supply the body with milk-based proteins, especially those which are predominantly Casein in nature like those contained within the Systemex.

In this French study originating from the Sainte-Marguerite Hospital in Marseille, a diet rich in Casein appears to actually protect subjects (non-obese mice who have a genetic predisposition for developing diabetes: NOD mice) from developing diabetes and then passing it on to their young. Specifically, Casein fed NOD female mice were protected against spontaneous diabetes and male NOD mice against acute Cyclosphosphamide or Cy-induced diabetes while also lessening the severity of insulitis.

Moreover, Casein has been found to exhibit the highest ratio of Total Essential Amino Acids to total Nitrogen of all foods and proteins reported by the FAO/WHO Expert Group.

The specific Amino Acid configuration of Casein just mentioned appears to effectively compete against the inflammatory cytokines response produced by the lymphocytes of the Immune System mentioned earlier, and may even reduce or circumvent the possibility of such an aberrant response, protecting beta cell integrity within the pancreas and the subsequent production of Insulin from amino acids derived from its structure. No other changes in the Immune System could account for these results. Such may also allow or ensure ß-cell "rest," a treatment strategy now of medical preference.

Interestingly, egg-based (albumin) proteins and other hydrolyzed proteins did not demonstrate this "protective" effect; Subjects fed albumin based proteins developed insulitis in 10 weeks. This is yet another strong reason for those who have a family history of diabetes, especially pregnant or lactating women and children, to supplement with Vaxa Systemex & TriCardia+. Because of the autoimmune nature of diabetes, supplementation with Immune-Aid+ is also recommended. Immune-Aid+ is formulated to ensure T-cell integrity and subsequent appropriate dispersal of those cellular populations throughout the bloodstream.

The good news is that with appropriate medical and nutritional support of the Immune System, and early detection, diabetes may be controlled and possibly even prevented.

References:

Arfeen S; Goodship TH; Kirkwood A; Channon S; Ward MK, "1% amino acid peritoneal dialysate: single-cycle study in diabetic patients with end-stage renal disease," Department of Internal Medicine, University of Missouri Health Sciences Center, Am J Kidney Dis 1994 Jan;23(1):86-90.

Arden SD; Roep BO; Neophytou PI; Usac EF; et al., "Imogen 38: a novel 38-kD islet mitochondrial autoantigen recognized by T cells from a newly diagnosed type 1 diabetic patient," Department of Clinical Biochemistry, University of Cambridge, Addenbrooke's Hospital, United Kingdom, J Clin Invest 1996 Jan 15;97(2):551-61.

Blackwood, A.L., M.D.; Manual of Materia Medica, Therapeutics and Pharmacology, Second Edition, Chicago, 1922. Birk OS; Douek DC; Elias D; Takacs K; et al., "A role of Hsp60 in autoimmune diabetes: analysis in a transgenic model, "Medical Research Council Clinical Sciences Centre, Royal Postgraduate Medical School, Hammersmith Hospital, London, United Kingdom, Proc Natl Acad Sci U S A 1996 Feb 6;93(3):1032-7.

Clark, John Henry, "Dictionary of Practical Materia Media," Ninth Edition, London, 1901. Dewey,W.A., M.D.; Practical Homeopathic Therapeutics, Third Edition, San Francisco, 1934.

Hancock WW; Polanski M; Zhang J; Blogg N; Weiner HL, "Suppression of insulitis in non-obese diabetic (NOD) mice by oral Insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-beta, and prostaglandin-E," Department of Pathology, New England Deaconess Hospital, Boston, Am J Pathol 1995 Nov;147(5):1193-9.

Hermitte L; Atlan-Gepner C; Payan MJ; Mehelleb M; Vialettes B, "Dietary protection against diabetes in NOD mice: lack of a major change in the Immune System," Service de Nutrition, Endocrinologie, Maladies metaboliques, Hospital Sainte-Marguerite, Marseille, France, Diabete Metab 1995 Oct; 21(4):261-8.

Hopkins BA; Rakes AH; Daniel TE; Zimmerman CA; Croom WJ Jr., "Effects of intraperitoneal L-leucine, L-isoleucine, L-valine, and L-arginine on milk fat depression in early lactation cows," Department of Animal Science, North Carolina State University, J Dairy Sci 1994 Apr;77(4):1084-92.

Huges, Richard, M.D., "Principles and Practice of of Homeopathy," fourth edition, London, 1901. Johnson-Tardieu JM; Walworth EW; Cornelius JG; Ye X; et al., "Autoimmune diabetes-prone NOD mice express the Lyt2 alpha (Lyt2.1) and Lyt3 alpha (Lyt3.1) alleles of CD8,"Department of Pathology & Laboratory Medicine, University of Florida College of Medicine, Immunogenetics 1996;43(1-2):6-12.

Kendrew, Sir John; "The Encyclopedia of Molecular Biology," Blackwell Publishers, Oxford, Oxfordshire, England, 1994.

Pieper GM; Jordan M; Adams MB; Roza AM, "Syngeneic pancreatic islet transplantation reverses endothelial dysfunction in experimental diabetes," Department of Transplant Surgery, Medical College of Wisconsin, Diabetes 1995 Sep;44(9):1106-13.

Ruilope LM, "Effects of angiotensin converting enzyme inhibitors on the progression of diabetic nephropathy." Unidad de Hipertension, Hospital 12 de Octubre, Madrid, Spain, J Hypertens Suppl 1995 Aug;13(2):S91-3.

Takacs K; Douek DC; Altmann DM, "Exacerbated autoimmunity associated with a T helper-1 cytokine profile shift in H-2E-transgenic mice," Clinical Sciences Centre, Royal Postgraduate Medical School, Hammersmith Hospital, London, UK, Eur J Immunol 1995 Nov; 25 (11):3134-41.

Zipris D; Greiner DL; Malkani S; Whalen B; et al., "Cytokine gene expression in islets and thyroids of BB rats. IFN-gamma and IL-12p40 mRNA increase with age in both diabetic and Insulin-treated nondiabetic BB rats," Department of Medicine, University of Massachusetts Medical Center, J Immunol 1996 Feb 1;156(3):1315-21.

 

Caution: For any suspected or known illness or dysfunction, always consult your physician for medical diagnosis and treatment first. Statements contained herein have not been evaluated by the Food and Drug Administration. Products mentioned herein are not intended to diagnose, treat, cure, or prevent any disease.